If you reside in a house or apartment, or you just would like to keep your home free of electromagnetic fields There are a variety of methods to reduce exposure. One of the easiest is to reduce the usage of your electronic devices. block emf is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another method to protect your home against EMF radiations is to put up an RF shielding cover. It's a kind of net that contains EMF shielding. It is utilized to prevent EMFs from entering a space. Another alternative is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Several studies have shown that the non-ionizing RF EMF has anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This could be the reason for the long-term responses seen in patients with advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not evident.
Aspects of AM RF EMFs on HCC tumor growth in vivo were examined in mice. The tumors were divided into 3 groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of tumour shrinkage.
The mechanism of cancer-specific AM RF EMF might be driven by the fact that tumour cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated by CACNA1H which is a protein that is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have more broader implications in the diagnosis and treatment of a variety of cancers.
emf blocker in the control group were not exposed EMF from radiofrequency, and fed a standard mouse diet. The tumours in the HCCMF group were treated with Huh7 cells at the time they were five-seven weeks old. The tumors were removed in cases of excessive burden.
The tumours in the three groups also displayed different growth curves. The HCCMF-treated tumors showed a significant decrease in tumour size after 8 weeks. However, the tumors which were treated by RCF didn't show shrinkage. The difference was significant. The tumors treated by RCF were able to show necrosis, which is typical in tumours exposed to RCF. There is a possibility that this necrosis was due to the lack of oxygen in the larger tumours.
In summary, the results show the fact that AM EMF is a powerful source of anti-cancer properties in vitro as well as in the vivo. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. It is possible that AM RF EMF triggers these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the growth of HCC tumors in living tissue.