Whether you live in an apartment or a house, or you just would like to ensure your home is free of EMFs, there are a number of methods to reduce exposure. One of the most effective is to restrict your electronic device use. You could also consider EMF blocker paint to block EMF radiation from reaching your home. emf blockers to protect your house from EMF radiation would be to install an RF shielding canopy. This is a fabric made of net that has EMF shielding. It's used to block EMFs from entering a room. Another option is to get your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This could explain the long-term responses seen in some patients with advanced HCC. However, the mechanism behind AM EMF's effects on cancer patients is not clear.
The effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were split into three groups. First, emf blocker that was unaffected to RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to the one used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on the tumours was compared to that of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is caused by CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H may have broader implications for diagnosis and treatment of various cancers.
The tumors in the controls were never exposed to EMF from RF, and fed a normal diet of mice. The tumors in those in the HCCMF group were injected with Huh7 cells when they were five-seven weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, the tumors treated with RCF didn't show shrinkage. The difference was significant. block emf treated by RCF had necrosis, which is typical in tumours exposed to RCF. There is a possibility that the necrosis is caused by the lack of oxygen in the more invasive tumours.
In conclusion, the findings suggest an AM-RF EMF is a powerful source of anticancer effects in vitro and in the vivo. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in living tissue.